Presentamos el caso de una paciente de 44 años estudiada por amenorrea e hiperprolactinemia. No refería galactorrea, cefalea ni alteraciones en la visión. HIPERPROLACTINEMIA Y PROLACTINOMA. MP Diagnóstico específico: PRL se deben medir en todo paciente con hipogonadismo o. A hiperprolactinemia causa hipogonadismo hipogonadotrófico principalmente por inibir a secreção pulsátil do GnRH, além de inibir diretamente a.

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Twenty-four hour secretory patterns of prolactin in women.

Prolactinomas are usually classified as microprolactinomas less than 1 cm or macroprolactinomas larger than 1 cmwhich can either be confined or invasive. Comparison of cabergoline and bromocriptine in patients with asymptomatic incidental hyperprolactinemia undergoing ICSI-ET. A amejorrea deve ser monitorada clinicamente a cada trimestre. Cabergoline Comparative Study Group.

Comparison of the effects of cabergoline and bromocriptine in women with hyperprolactinemic amenorrhea. Emotional aspects of hyperprolactinemia.

Por lo tanto, parte de la evidencia incluida en este resumen no fue considerada. Se houver um crescimento significativo do tumor, deve-se reintroduzir o AD.

Osteocalcin levels in patients with microprolactinoma before and during medical treatment. However, it is not clear if this translates into clinical benefits. Ribeiro RS, Abucham J. Radiotherapy for hiperprolacinemia pituitary tumors. Insulin sensitivity and lipid profile in prolactinoma patients before and after normalization of prolactin by dopamine agonist therapy.


[Current diagnosis and treatment of hyperprolactinemia].

N Engl J Med. Hyperprolactinemia is a frequent neuroendocrinological condition that should be approached in an orderly and integral fashion, starting with a complete clinical history.

Do the limits of serum prolactin in disconnection hyperprolactinaemia need re-definition? Outcomes of transsphenoidal surgery in prolactinomas: Pakistan Journal of Medical Sciences Online. Cabergoline or bromocriptine for prolactinoma?.

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How to cite this article. Potential cardiac valve effects of dopamine agonists in hyperprolactinemia. Increased prevalence of subclinical opr valve fibrosis in patients with prolactinomas on long-term bromocriptine and cabergoline treatment.

BMI and metabolic profile in patients with prolactinoma before and after treatment with dopamine agonists.

Human macroprolactin displays low biological activity via its homologous receptor in a new sensitive bioassay. Dopamine as a prolactin PRL inhibitor.

Recurrence of hyperprolactinemia after withdrawal of dopamine agonists: Body fat in men with prolactinoma. Diagnosis and treatment of yperprolactinemia: J Clin Endocrinol Metab.


Hyperprolactinemia causes hypogonadism, menstrual irregularities or amenorrhea in women, low serum testosterone levels in men, and infertility and sexual dysfunction in both men and women. Once physiological causes such as pregnancy, systemic disorders such as primary hypothyroidism and the use of drugs with dopamine antagonistic actions such as metochlopramide have been ruled out, the most common cause of hyperprolactinemia is a PRL-secreting pituitary adenoma or prolactinoma.

Temozolomide in the management of dopamine agonist-resistant prolactinomas.


Bone marker and bone density responses to dopamine agonist therapy in hyperprolactinemic males. High prevalence of radiological vertebral fractures in women with prolactin-secreting pituitary adenomas.

A comparison of cabergoline and bromocriptine in the treatment of hyperprolactinemic amenorrhea. Cabergoline is a long-acting dopamine receptor agonist which might offer advantages over hiperprolactinemix.